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CT Head 8 min read

Recognizing intracranial hemorrhage

Learning objectives

  • Differentiate the five compartments of intracranial blood
  • Track how blood density evolves with time
  • Report the modifiers that change management

Step-by-step approach

  1. 1

    Density and time

    Hyperacute-to-acute blood is bright; over 1–2 weeks it becomes isodense to brain, then hypodense. A subacute isodense subdural is a classic trap.

  2. 2

    Extradural

    Lentiform (lens-shaped), limited by sutures, usually arterial and post-traumatic — often with an overlying fracture.

  3. 3

    Subdural

    Crescentic, crosses sutures but not dural reflections; common in older patients and may follow trivial trauma.

  4. 4

    Subarachnoid

    Blood in sulci and cisterns. Non-traumatic SAH means aneurysm until proven otherwise.

  5. 5

    Intraparenchymal and intraventricular

    Location suggests cause: basal ganglia favours hypertension; lobar in older patients suggests amyloid; always note intraventricular extension.

  6. 6

    The four modifiers

    Every hemorrhage report needs: location, size, intraventricular extension, and midline shift/mass effect.

Common mistakes

  • Missing isodense subacute subdurals
  • Not switching windows to separate blood from adjacent skull
  • Omitting mass-effect assessment

Summary checklist

  • Which compartment?
  • Acute or evolving?
  • IVH?
  • Midline shift?
  • Cisterns open?

Ask RADSpace about this guide

AI responses are for education and may contain errors. RADSpace is not for primary clinical diagnosis.